Scientists identify genetic locations implicated in development of frontal fibrosing alopecia.
Frontal fibrosing alopecia (FFA) is an increasingly common hair disorder characterised, in most cases, by a specific pattern of hair loss along the frontal hairline. The exact cause of this condition is complex and likely to be related to both genetic and environmental factors (i.e. multifactorial). A recent exciting new study published in the Journal of Investigative Dermatology (November 2017) looked at how some of the genetic factors might contribute to this disease. In particular, this study
The Hair Loss PSP is asking everyone whose life is affected by hair loss and people involved in treating hair loss to think about the prevention, diagnosis and treatment of hair loss problems by asking, “What questions do you have that need to be answered by research?
In a recent edition of Nature an exciting new approach to managing alopecia areata was discussed. The research team demonstrated the crucial role of a particular white blood cell, known as the cytotoxic CD 8 T lymphocyte, more specifically the CD8 NKG2D T Cell.
In a mouse model it was demonstrated that this cell promotes the development of alopecia areata and leads to the autoimmune attack on the hair follicle. It is likely that the CD8 cells produce Interferon gamma which leads to collaps
Hair loss (alopecia) is a common problem that has been shown to have a significant impact on psychological well-being and quality of life, and can sometimes also signify an underlying medical problem. Treatment of hair loss disorders is often challenging due to a number of factors, including limited understanding of the natural history of the condition, poor disease definitions, lack of validated severity scales and generally poor quality of evidence for treatment. Only a limited number of hair